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Objective: To explore the repair effect of Wnt3a on injured spinal cord in rats. Methods: Moderate spinal cord contusion injury was made in 40 adult Sprague-Dawley rats at T10. Fifte. Publication » Activation of Wnt3a signaling stimulates...

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  • Objective: To explore the repair effect of Wnt3a on injured spinal cord in rats. Methods: Moderate spinal cord contusion injury was made in 40 adult Sprague-Dawley rats at T10. Fifte.

  • Publication » Activation of Wnt3a signaling stimulates intestinal epithelial repair by prom. functional organ to replace native organ lack or loss. Short bowel syndrome (SBS) is a li.

  • . Light and electron microscopic works showed that the Wnt3a-treated group had moderate repair effect of myelin and axons. Immunohistochemical analysis showed a significant .

  • of calcium sulfate cement for bone repair by dicalcium silicate. Journal of Alloys and Com. Hayashi, M, Nakashima, T, Taniguchi, M (2012) Osteoprotection by semaphorin 3A. Na.

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  • while TDG inhibits the methylation activity of Dnmt3a in vitro. These data suggest a mechanistic link between DNA repair and remethylation at sites affected by methylcytosine dea.

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  • We show that mammalian O3a also functions at the G2 to M checkpoint in the cell cycle and triggers the repair of damaged DNA. By gene array analysis, O3a was found.

  • RESULTS:CelecoxibtreatmentofMin/?micereducedgranula-tiontissuethicknesscellproliferationangiogenesisandcollagenproductionrelativetocontrols.CONCLUSIO.

  • .Furthermore, functional interactions with the DNA-methyltransferase Dnmt3a were found. and this interaction stimulates the glycosylase activity, allowing for the repair of the T/G m.

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  • of repair are many, but one possible reason is the lack of migration of oligodendrocyte precursor cells to the lesion. The guidance molecules Semaphorin 3A and 3F, already know.

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